A frequent phenomenon is that when one half of a couple that’s been together for a very long time expires, another also dies shortly after. The usual, intimate, explanation is that they loved each other so much that they couldn’t live without each other. He or she died from a’broken heart’, ‘couldn’t live ‘, ‘was dependent on their partner’.
Is there a correlation between the death of one spouse and the subsequent death of another soon afterwards? The theory is that the desire to live may have a specific influence on the immune system. The brain that’s in charge of our body might have a shut-down mechanism, which can be triggered in certain instances. Those cases are like fatal accidents or specific illnesses, where the mind knows it will not have the ability to cope.
This mechanism may control ‘suicide cells’. Lately, suicide cells what scientists define as programmed cell death (PCD) has formed the foundation for continuing biogenetic research. PCD is the passing of a cell that’s mediated by an intracellular programme. There are three significant kinds of PCDs. Type I cell death is called apoptosis. Type II is autophagic and Type III is necrotic cell-death. Cells can be murdered by injurious agents or be instructed to commit suicide.
Tenete a mente
If there’s a threat to the integrity of an organism by certain cells, PCD is required to destroy those cells. Typical examples of these cases are: cells that are infected by viruses, DNA damage, cells of the immune system and cancer cells. In certain kinds of cancer cells apoptosis is triggered by radiation or substances used for treatment. What makes a cell opt to commit suicide? The writer considers that it is the imbalance between positive and negative signals sent by the mind. If there’s a scarcity of the positive signs (no desire to live) necessary for survival or negative signals are routed significance’no desire to continue to live’, the shut-down mechanism may be triggered.
There have been many reports of instances where patients recovered after clearly being diagnosed with cancer. This phenomenon might be explained by the stimulation of the PCD mechanism by’desire to live’ positive signals. Sometimes, viruses that are associated with cancers can use tricks, like creating a protein that inactivates the apoptosis signal. In these cases the cancer cells won’t only continue to live and proliferate, but they will become more resistant to apoptosis.
Further comprehension of these tricks and decoy molecules generated to protect cancer cells would enable researchers to reactivate and conquer those protective tricks to be able to destroy cells that are dangerous. The author also believes that future research on removing and destroying cancerous cells may be implemented in two stages. The first phase is to distinguish, identify and mark cancerous cells. In phase two the goal is to trigger suicide cells in the chosen region or set of cells and skip the present security of the cancer cells. Strong psychological and family support is critical for such a restoration. Moreover, the individual must believe in and hope for a healthy and bright future.